左心室壁见大片陈旧性透壁性梗死。尽管心肌壁的厚度正常，但内部仅为薄弱纤维。由于梗死范围太大，以至愈后心室壁被胶原纤维所取代，形成动脉瘤。动脉瘤弹性差，降低了心脏的每搏排出量并增大了残余心肌张力。
There has been a previous extensive transmural myocardial infarction involving the free wall of the left ventricle. Note that the thickness of the myocardial wall is normal superiorly, but inferiorly is only a thin fibrous wall. The infarction was so extensive that, after healing, the ventricular wall was replaced by a thin band of collagen, forming an aneurysm. Such an aneurysm represents non-contractile tissue that reduces stroke volume and strains the remaining myocardium. The stasis of blood in the aneurysm predisposes to mural thrombosis.